UNFUCG
DashboardSearchChatBookmarksNotificationsActivityPremiumProfile
?
Home
Search
Chat
Saved
Profile
Episode
If I Had 5lbs of Visceral Fat to Lose, Here’s Exactly What I’d Do (Step-by-Step)
~58 min
Episode Brief·YouTube

If I Had 5lbs of Visceral Fat to Lose, Here’s Exactly What I’d Do (Step-by-Step)

Thomas DeLauer
Watch on YouTube Add to chat My bookmarks← All sources

TL;DR

The four things you'd lose by not watching

4 items

TL;DR

The four things you'd lose by not watching

4 items
1

Lengthen the time between meals to 5–6 hours without snacking to restore insulin sensitivity, even without cutting calories.

2

Combine Norwegian 4×4 high-intensity intervals (2–3×/week) with low-intensity rucking or hill walking (2–4×/week) to both stress and metabolically train the liver to oxidize fat.

3

Add omega-3s and C15 saturated fat (from aged cheese or the supplement Fatty15) to improve liver fatty acid oxidation and protect liver cells from ferroptosis.

4

Manage cortisol by reducing life stress, using sauna or hot baths, and supporting GABA with glutamine or 5-HTP, because visceral fat has 4× the cortisol receptors of other fat.

Protocols

Concrete recipes — what, when, how much, and why

9 items

Lengthen Time Between Meals

WhatIncrease the gap between meals to 5–6 hours without any caloric intake (not even coffee with cream), without necessarily changing total calories or fasting.
WhenDaily, starting with breakfast and then waiting 5–6 hours before the next meal.
Dose5–6 hours between meals; no snacking, no caloric beverages.
For whomAnyone with suspected insulin resistance or visceral fat, especially those who find carbohydrate reduction difficult.
WhyProlonged periods without insulin spikes allow cells to restore insulin sensitivity, breaking the cycle of hyperinsulinemia that drives insulin resistance and fatty liver.
CaveatsNot necessarily full intermittent fasting; just spacing meals. If you can also reduce carbs, do it, but spacing is the primary lever.

DeLauer argues that insulin resistance is largely a result of constant insulin signaling, which desensitizes cells. By creating longer windows without food, the body can resensitize to insulin. He references intermittent fasting and alternate-day fasting research showing that even without calorie reduction, insulin sensitivity improves. He emphasizes that this is the first and easiest step, and that Dr. Sha Omera, a visceral fat expert, agrees with this approach. He notes that many people struggle with carb reduction, so meal spacing is a more accessible starting point.

Mechanism

Hyperinsulinemia causes cells to downregulate insulin receptors. Extended periods without food lower insulin levels, allowing cells to upregulate receptors and become more responsive. This reduces the drive for liver fat accumulation and subsequent visceral fat expansion.

I don't even care at first if we change our calorie count. Okay? Increase the time between meals and sporadically have longer periods of time.

Also said
“Remember the insulin resistance is a result of insulin hyperinsulinemia. Insulin constantly pumping and yelling at the cell to the point where the cell just turns off its interest.”— Vivid metaphor for insulin resistance.
“People will say, 'Hey, reduce your carbohydrate intake.' You can definitely do that, too. That can improve insulin resistance tremendously. But I find that a lot of times that is a challenge for people.”— Positions meal spacing as an alternative or complement to carb reduction.

Norwegian 4×4 Interval Training

WhatFour minutes of high-intensity exercise followed by three minutes of active recovery, repeated four times (total 28 minutes).
When2–3 times per week, on separate days from low-intensity cardio or as part of a split routine.
Dose4 minutes on, 3 minutes off, 4 rounds. Intensity should leave you exhausted at the end of each 4-minute interval.
For whomAnyone who can handle high-intensity exercise biomechanically and from a CNS standpoint; if not, shorter intervals (30–60 seconds) can be used.
WhyImproves VO2 max, creates a stress response that mobilizes visceral fat, and enhances metabolic health.
CaveatsIf you cannot handle the 4-minute duration, start with shorter intervals. Ensure proper recovery between sessions.

DeLauer presents the Norwegian 4×4 as a research-backed protocol that maximizes VO2 max improvement, which is strongly correlated with lower cardiovascular disease and better metabolic health. He notes that Dr. Sha Omera recommends sprinting for visceral fat loss, and this protocol fits that high-intensity need. The 4-minute work period is long enough to create significant metabolic stress but not so short that it overly taxes the nervous system. He suggests doing it on a bike, elliptical, running, or jump rope—anything that allows sustained high effort. He also mentions that if biomechanics or CNS fatigue are issues, shorter intervals like 1 minute on/off or 30 seconds on/off are acceptable alternatives.

Mechanism

High-intensity intervals trigger a stress response that liberates fatty acids from visceral fat stores. The repeated bouts also improve mitochondrial efficiency and VO2 max, which enhances overall fat oxidation capacity.

I would do four minutes on, three minutes off. And I would do that four times. So four on, three off, four on, three off, four on, three off, four on, done.

Also said
“At the end of that four minutes, I should be exhausted. Nice thing about this, you can do it running, you can do it biking, you can do it on elliptical, you can do it jumping rope, whatever is going to get you to that point.”— Specifies the intensity and flexibility of the protocol.

Low-Intensity Rucking or Hill Walking

What45 minutes of low-intensity steady-state cardio, such as walking with a weighted backpack (rucking) or walking uphill, keeping heart rate in zone 2.
When2–4 days per week, ideally on days not doing high-intensity intervals.
Dose45 minutes per session.
For whomAnyone, especially those with fatty liver or visceral fat; not intended to turn someone into an endurance athlete.
WhyUpregulates PPAR-alpha, which trains the liver to oxidize fatty acids, complementing the fat-liberating effect of high-intensity work.
CaveatsAvoid becoming a distance runner, as that may promote visceral fat storage. Keep intensity low and duration moderate.

DeLauer distinguishes this from the high-volume endurance training that Dr. Omera warns against. He explains that low-intensity activity specifically upregulates PPAR-alpha, a nuclear receptor that enhances hepatic fat oxidation. This is a metabolic adaptation that high-intensity work alone does not fully provide. He suggests rucking (walking with a backpack) or hill walking as practical, joint-friendly options. The combination of high-intensity intervals and low-intensity steady-state creates a dual-pronged attack: stress-induced mobilization and metabolic training of the liver.

Mechanism

Low-intensity exercise activates AMPK and PPAR-alpha pathways, increasing the expression of enzymes involved in fatty acid oxidation in the liver. This shifts the liver's metabolism toward burning fat rather than storing it, directly reducing liver fat and, by extension, visceral fat.

I'm asking you to put a little backpack on and go for a walk or go for a walk up a hill for 45 minutes because we need two systems to work.

Also said
“We need the metabolic shift of training the liver to oxidize fats and upregulating what's called PPAR.”— Names the specific pathway targeted by low-intensity cardio.

Omega-3 Supplementation for Liver Fat Oxidation

WhatTake a high-quality omega-3 supplement to improve PPAR-alpha activity and reduce liver fat.
WhenDaily, with meals.
DoseNot specified; follow supplement label.
For whomAnyone with fatty liver or visceral fat.
WhyOmega-3s increase fatty acid oxidation in the liver and decrease liver fat, as shown in a study published in Gastroenterology Research and Practice.
CaveatsNone mentioned.

DeLauer cites a specific study where omega-3 supplementation led to increased hepatic fatty acid oxidation and decreased liver fat, along with reduced GGT (a marker of liver stress). He positions this as the nutritional counterpart to low-intensity cardio, both working through PPAR-alpha. He does not specify a brand but emphasizes quality.

Mechanism

Omega-3 fatty acids activate PPAR-alpha, a transcription factor that upregulates genes involved in fatty acid uptake and beta-oxidation in the liver. This reduces lipid accumulation and liver stress.

Omega-3s, like a good quality omega-3 supplement, is going to improve what's called PPAR alpha, just like the low intensity cardio is going to do.

Also said
“This was published in gastroenterology research and practice. It's a very cool study that showed that people that added omega-3s in ended up increasing their fatty acid oxidation in the liver.”— Provides a specific study reference.

C15 Intake via Aged Cheese or Fatty15 Supplement

WhatConsume 1–2 ounces of pecorino romano or parmesan cheese daily, or take the Fatty15 supplement, to provide C15 saturated fat that protects liver cells from ferroptosis.
WhenDaily, with meals.
Dose1–2 ounces of cheese or as directed on Fatty15 supplement.
For whomAnyone with fatty liver or visceral fat; also for general cellular health.
WhyC15 stabilizes cell membranes, prevents oxidative damage and ferroptosis in liver cells, reducing inflammation and fatty liver.
CaveatsCheese option adds calories and saturated fat; supplement is a purified form. DeLauer has an affiliate relationship with Fatty15.

DeLauer explains that C15 is a saturated fat found in Mediterranean cheeses that has been studied by Dr. Van Watson. It prevents ferroptosis, an iron-dependent cell death that contributes to liver inflammation. By protecting liver cells, C15 reduces the inflammatory cascade that leads to fatty liver and visceral fat. He offers two routes: dietary cheese or the supplement Fatty15, which he has a discount code for. He also recommends Dr. Van Watson's book for those wanting to dive deeper. He emphasizes that this is a highly important intervention.

Mechanism

C15 incorporates into cell membranes, increasing their resistance to lipid peroxidation. This prevents ferroptosis, a form of cell death driven by iron and lipid peroxides, which is implicated in liver damage and inflammation. By preserving hepatocyte integrity, C15 reduces the inflammatory signals that promote fatty liver and visceral fat accumulation.

These Mediterranean cheeses contain a type of saturated fat that we've seen in the literature now called C15 that actually supports and protects the cellular structure within the liver also in a lot of cells and it prevents something called ferroptosis.

Also said
“I don't care if you get aged cheese or you use a supplement, but fatty 15 is a really cool supplement.”— Gives flexibility in sourcing C15.

Hot Bath Immersion for Lipolysis

WhatTake a hot bath immersed up to the neck to trigger heat shock proteins and growth hormone release, enhancing fat oxidation.
WhenAs desired, possibly on rest days or evenings.
DoseNot specified; presumably 15–30 minutes at a comfortably hot temperature.
For whomAnyone, especially those who cannot do sauna.
WhyHeat stress increases heat shock proteins, which alter fat utilization and oxidation rates, and boosts growth hormone, which promotes lipolysis.
CaveatsNone mentioned, but avoid if contraindicated (pregnancy, cardiovascular issues).

DeLauer mentions that he discussed this with Dr. Sha Omera. Heat exposure acts as a hormetic stressor, similar to exercise, triggering heat shock proteins that act as chaperones and modify how the body oxidizes fats. Additionally, the rise in growth hormone from heat stress (also seen with fasting and resistance training) increases lipolysis, liberating fat from stores. He positions this as a passive intervention that complements active training.

Mechanism

Heat shock proteins facilitate proper protein folding and cellular repair, but also influence metabolic pathways. Heat stress increases growth hormone secretion, which stimulates hormone-sensitive lipase, leading to greater release of free fatty acids from adipose tissue, including visceral fat.

Take a hot bath immersed up to your neck. Heat is going to trigger a stress response. It's actually going to allow for more fat to be oxidized.

Also said
“Fasting, taking breaks from food, sauna and resistance training all increase growth hormone levels which increase lipolysis which means it's allowing more fat to be liberated from these areas.”— Connects growth hormone to fat liberation.

Stress Reduction and Parasympathetic Activation

WhatEngage in enjoyable physical activities, use sauna, and consider supplements like 5-HTP, nicotinyl GABA (Troscriptions TrollM), or glutamine to shift toward parasympathetic tone and reduce cortisol-driven visceral fat storage.
WhenDaily stress management; supplements as needed (5-HTP short-term due to tolerance).
Dose5-HTP: low dose, short cycles; glutamine: dietary and occasional supplementation; TrollM: as directed.
For whomAnyone with high stress or suspected high visceral fat.
WhyHigh cortisol increases visceral fat storage due to dense glucocorticoid receptors; reducing stress and enhancing GABAergic tone lowers cortisol and shifts fat partitioning away from visceral depots.
Caveats5-HTP builds tolerance quickly; some debate whether it crosses the blood-brain barrier. Yohimbine (mentioned elsewhere) should only be used with exercise. MOTS-c peptide requires doctor consultation.

DeLauer emphasizes that stress is a major driver of visceral fat because visceral adipose tissue has four times the cortisol receptors of subcutaneous fat. Even with exercise, if you are chronically stressed, excess calories will be directed there. He recommends finding fun physical activities (like ultimate frisbee) to reduce stress. He personally uses 5-HTP for sleep and reduced reactivity, though he notes rapid tolerance. He also mentions nicotinyl GABA (Troscriptions TrollM) and glutamine to support GABA production, as GABA promotes parasympathetic tone. The goal is to live in fight-or-flight only when necessary and parasympathetic the rest of the time, creating a clear on/off switch rather than a constant gray area of stress.

Mechanism

Cortisol binds to glucocorticoid receptors, which are highly expressed in visceral fat, promoting lipoprotein lipase activity and fat storage. Reducing cortisol through stress management and enhancing GABA (which inhibits the stress response) decreases this signal. Glutamine is a precursor to GABA, and 5-HTP may increase serotonin, which can improve sleep and mood, indirectly lowering cortisol.

Personal experience

DeLauer shares that he personally uses 5-HTP and finds it helps him sleep and become less reactive, but he builds tolerance quickly and must cycle it or increase the dose.

The more stressed you are, the more likely you are to store fat in that visceral region itself. Okay? The body's just going to reallocate because you have more glucocorticoid receptors there.

Also said
“I am a fan of 5HTP. Some people say it doesn't actually cross the blood-brain barrier. My personal anecdotal experience with 5HTP is I build a tolerance with it very fast, so I can't use it for long or I have to increase the dose. But it does help me sleep and it helps me become less reactive.”— Personal anecdote with caveats.
“The more that you can influence this on or off versus a gray area all the time, the better the chance you have at burning through that visceral fat.”— Frames stress modulation as a binary switch rather than constant management.

Increase Polyphenol Intake

WhatDramatically increase polyphenols from berries, onions, garlic, and sulfur-containing vegetables, and consider extracts, to reduce inflammation and support liver health.
WhenDaily, with meals.
DoseNot specified; emphasize variety and consistency.
For whomAnyone, especially those with fatty liver or high inflammation.
WhyPolyphenols are anti-inflammatory and act as prebiotic fibers, supporting gut health and reducing systemic inflammation that drives fatty liver and visceral fat.
CaveatsNone mentioned.

DeLauer suggests that in the first 30 days of a visceral fat loss protocol, he would dramatically increase polyphenol intake. He mentions berries, onions, garlic, and sulfur-containing foods because they support liver detoxification pathways. Polyphenols also serve as prebiotics, improving gut health, which in turn reduces systemic inflammation. He ties this to the overall goal of improving liver health to reverse fatty liver and visceral fat.

Mechanism

Polyphenols scavenge free radicals and modulate inflammatory pathways like NF-kB. They also feed beneficial gut bacteria, producing short-chain fatty acids that strengthen the gut barrier and reduce endotoxin-driven liver inflammation.

The polyphenols not only are a prebiotic fiber that help us out, but they are something that are truly anti-inflammatory.

Also said
“I might eat things like onions and garlic and the things that are sulfur containing that I've talked about because it helps the liver, helps support the liver, and that's going to be very good.”— Specifies sulfur-containing foods for liver support.

High Water Intake for Thermogenesis and Lymphatic Drainage

WhatDrink a large amount of water throughout the day to increase resting energy expenditure and improve lymphatic clearance.
WhenThroughout the day, consistently.
DoseNot specified; 'a ton of water' without flushing minerals.
For whomEveryone, but especially those with fatty liver or toxin exposure.
WhyWater-induced thermogenesis can raise metabolic rate by up to 25% for 60 minutes, and hydration supports lymphatic drainage, reducing liver burden.
CaveatsDon't overhydrate to the point of mineral depletion.

DeLauer cites evidence that water consumption increases resting energy expenditure by up to 25% for up to an hour, due to the energy cost of adjusting osmotic gradients. He also notes that water helps flush toxins and microplastics from tissues via lymphatic drainage, which reduces the detoxification load on the liver. He ties this to a previous video on microplastics. He emphasizes that hydration is a simple, effective tool for fat loss and liver health.

Mechanism

Water ingestion triggers a thermogenic response as the body works to maintain osmotic balance, particularly involving sodium gradients. Additionally, adequate hydration supports lymph flow, which removes metabolic waste and reduces inflammatory load on the liver.

There is now evidence that supports that drinking water can increase the resting energy expenditure for up to 25% up to 25% for up to 60 minutes after consumption.

Also said
“Hydration does increase fat loss and it has to do with osmolality. has to do with sort of uh changing the thermo regulation that's occurring there when you're absorbing fluid and having to change the gradient on the osmolality with sodium.”— Explains the thermogenic mechanism.

What's new

Personal practice updates, fresh positions, predictions

5 items

visceral-fat-as-liver-fat-proxy

early in the video

DeLauer argues that visceral fat is almost always preceded by fatty liver and insulin resistance, so treating the liver is the key to losing visceral fat.

Why this matters: Reframes visceral fat loss as a liver health problem rather than a simple calorie deficit issue, shifting the focus to hepatic fat oxidation and inflammation.

Background

Mainstream advice often targets overall body fat without distinguishing visceral fat. DeLauer, citing Dr. Sha Omera, says MRI scans show visceral fat expansion stems from fatty liver, which itself stems from insulin resistance and inflammation.

DeLauer explains that people often dismiss fatty liver but acknowledge visceral fat, yet the two are tightly linked. He describes visceral fat as an inflammatory organ system that develops its own life, creating a vicious cycle: inflammation drives insulin resistance, which drives more liver fat, which expands into visceral fat. Therefore, interventions must break this cycle at the liver level, not just create a caloric deficit. He emphasizes that even metabolically healthy people have some visceral fat, but the goal is to keep it limited by improving liver health and insulin sensitivity.

Visceral fat is more than likely an additional proxy for liver fat. What does that mean? It means that it's likely starting as a fatty liver and it's expanding around our organs.

Also said
“A fatty liver stems more than likely from insulin resistance. And one other thing, inflammation, right? And inflammation is often is often times at the root of insulin resistance.”— Connects the chain from inflammation to insulin resistance to fatty liver to visceral fat.
“We have to realize that we're always going to have a certain degree of fat around the liver and probably a certain degree of even, you know, visceral fat. We just want to keep it limited as much as possible.”— Normalizes the presence of some visceral fat while emphasizing the goal of limiting it.

dual-training-approach-for-visceral-fat

after introducing meal spacing

DeLauer recommends both high-intensity intervals (Norwegian 4×4) and low-intensity steady-state (rucking/walking) to address visceral fat through two distinct mechanisms: stress-induced mobilization and PPAR-alpha upregulation.

Why this matters: Pushes back against the idea that only high-intensity or only low-intensity is best; he argues both are needed for different biological pathways.

Background

Dr. Sha Omera advocates sprinting and warns that distance runners store more visceral fat. DeLauer agrees with the sprinting part but adds that low-intensity work is necessary to train the liver to oxidize fats via PPAR-alpha, not to become an endurance athlete.

DeLauer explains that the body adapts to whatever stimulus it receives, so altering training is crucial. High-intensity work like Norwegian 4×4 creates a stress response that liberates visceral fat, while low-intensity zone 2 cardio upregulates PPAR-alpha, which enhances the liver's ability to oxidize fatty acids. He cautions against becoming a distance runner, as that may promote visceral fat storage, but a 45-minute weighted walk or hill walk is sufficient. He suggests 2–3 days of Norwegian 4×4 and 2–4 days of low-slow rucking or hill walking per week.

We need the visceral fat to be burned and activated through the stress response of the high-intensity activity, but we need the metabolic shift of training the liver to oxidize fats and upregulating what's called PPAR.

Also said
“I'm not asking you to become an endurance or distance runner. I'm asking you to put a little backpack on and go for a walk or go for a walk up a hill for 45 minutes.”— Clarifies the low-intensity prescription is not about becoming a runner but about gentle, consistent movement.
“Dr. Sean Omera says, 'Hey, distance runners have higher amounts of visceral fat because as a as a uh sort of a storing system for that activity, they store visceral fat.' He's not incorrect, although it's somewhat speculative.”— Acknowledges the counterpoint while still advocating for low-intensity work.

c15-saturated-fat-for-liver-protection

after discussing omega-3s

DeLauer highlights C15, a saturated fat found in aged Mediterranean cheeses and available as the supplement Fatty15, as a compound that protects liver cells from ferroptosis and reduces liver fat.

Why this matters: Introduces a specific, lesser-known fatty acid as a targeted intervention for liver health, moving beyond generic 'healthy fats' advice.

Background

C15 (pentadecanoic acid) has been studied by Dr. Van Watson, who argues that humans are deficient in it. DeLauer connects it to preventing ferroptosis, an iron-dependent form of cell death that contributes to liver inflammation and fatty liver.

DeLauer explains that C15 supports the cellular structure of liver cells, preventing them from becoming oxidized and undergoing premature death (ferroptosis). This reduces liver inflammation, which in turn reduces fatty liver and visceral fat accumulation. He suggests either consuming 1–2 ounces per day of pecorino romano or parmesan cheese, or taking the Fatty15 supplement. He discloses an affiliate link for Fatty15 and mentions having interviewed the researcher behind it. He also recommends Dr. Van Watson's book for deeper understanding.

These Mediterranean cheeses contain a type of saturated fat that we've seen in the literature now called C15 that actually supports and protects the cellular structure within the liver also in a lot of cells and it prevents something called ferroptosis.

Also said
“Fatty 15 is the isolated form of the C-15 saturated fat. So, it makes the cell have structure so it doesn't become oxidized and ultimately lead to premature cellular death within the liver which leads to guess what? Inflammation and more fatty liver and visceral fat.”— Directly links C15 supplementation to the chain of liver health and visceral fat reduction.

cortisol-receptors-in-visceral-fat

mid-video, after hot baths

DeLauer states that visceral fat has four times as many glucocorticoid (cortisol) receptors as other fat, making stress a disproportionately powerful driver of visceral fat storage.

Why this matters: Provides a concrete physiological reason why stress management is not just wellness fluff but a direct lever for visceral fat loss.

Background

The link between stress and belly fat is often discussed vaguely. DeLauer quantifies it: four times the receptor density means insulin and cortisol have a much stronger impact on visceral fat than elsewhere.

He explains that when cortisol is high, the body preferentially stores fat in the visceral region because of the high density of glucocorticoid receptors there. Even if you exercise, if you are chronically stressed, any overeating or metabolic surplus will be directed to visceral fat. Conversely, when relaxed, the body is more likely to burn from visceral fat stores. He recommends finding enjoyable physical activities, using sauna, and supplements like 5-HTP or glutamine to shift the nervous system toward parasympathetic tone, thereby reducing the signal for visceral storage.

When you have high amounts of stress and cortisol, you're going to have four times as many cortisol receptors in the visceral fat, which means that insulin and cortisol, all these things are going to have a stronger impact on the visceral fat than they would anywhere else in the body.

Also said
“The more stressed you are, the more likely you are to store fat in that visceral region itself. Okay? The body's just going to reallocate because you have more glucocorticoid receptors there.”— Reinforces the reallocation concept.

water-induced-thermogenesis

near the end

DeLauer claims that drinking water can increase resting energy expenditure by up to 25% for up to 60 minutes after consumption, aiding fat loss.

Why this matters: Quantifies a simple, zero-cost intervention with a specific metabolic effect, making hydration a more active tool than usually portrayed.

Background

Hydration is typically recommended for general health, not as a direct fat-loss lever. DeLauer cites evidence that water consumption triggers a thermogenic effect due to osmolality changes and thermoregulation.

He explains that the body must adjust the osmotic gradient when absorbing water, which requires energy and raises metabolic rate. He also notes that water improves lymphatic drainage, helping clear waste and reducing the burden on the liver. He cautions against overhydration that could flush out minerals, but emphasizes that adequate water intake is a simple, effective strategy. He ties it to a previous video on microplastics, where water helped flush toxins from tissues.

There is now evidence that supports that drinking water can increase the resting energy expenditure for up to 25% up to 25% for up to 60 minutes after consumption.

Also said
“Hydration does increase fat loss and it has to do with osmolality. has to do with sort of uh changing the thermo regulation that's occurring there when you're absorbing fluid and having to change the gradient on the osmolality with sodium.”— Provides a mechanistic hint for the thermogenic effect.

Recommendations

Products, supplements, and tools mentioned in the episode

7 items

Omega-3 Supplement

Supplement

A high-quality omega-3 supplement to improve PPAR-alpha activity and reduce liver fat.

DeLauer does not specify a brand but emphasizes quality. He cites a study from Gastroenterology Research and Practice showing increased fatty acid oxidation and decreased liver fat with omega-3 supplementation.

Omega-3s, like a good quality omega-3 supplement, is going to improve what's called PPAR alpha, just like the low intensity cardio is going to do.

Find Omega-3

Yohimbine

Supplement

A beta agonist that liberates fat, used at 2–5 mg, but only if committed to exercise.

DeLauer suggests yohimbine as a potential aid for visceral fat loss because it acts as a beta agonist, increasing lipolysis. However, he strongly cautions that it should only be used if the person is going to exercise, both to avoid jitters and to ensure the liberated fats are actually oxidized. He does not recommend it for sedentary individuals.

Yohimbeine because yohim bean even though it gives some people some jitters in a low dose like 2 to 5 milligrams, it really is a powerful beta agonist. So, you're essentially making it so that you liberate more fat, which can happen across the body, but also can happen at the liver level.

Also said
“Caveat, I do not recommend taking this unless you can commit to exercise. A, you just be jittery. B, it's going to work way, way better, and you're going to train your body to utilize those fats better.”— Critical caveat for safe use.
Find Yohimbine

5-HTP

Supplement

A serotonin precursor that DeLauer uses personally for sleep and reduced reactivity, despite rapid tolerance buildup.

DeLauer shares his personal experience with 5-HTP, noting that while some question its ability to cross the blood-brain barrier, he finds it effective for sleep and emotional regulation. He warns that he builds tolerance quickly and must cycle it or increase the dose. He recommends it as a short-term tool for stress reduction, which indirectly helps visceral fat loss by lowering cortisol.

Personal experience

DeLauer says: 'My personal anecdotal experience with 5HTP is I build a tolerance with it very fast, so I can't use it for long or I have to increase the dose. But it does help me sleep and it helps me become less reactive.'

I am a fan of 5HTP. Some people say it doesn't actually cross the blood-brain barrier. My personal anecdotal experience with 5HTP is I build a tolerance with it very fast, so I can't use it for long or I have to increase the dose. But it does help me sleep and it helps me become less reactive.

Find 5-HTP

Troscriptions TrollM

Product

A product containing nicotinyl GABA, kava, and other compounds to influence the GABA system for relaxation.

DeLauer mentions Troscriptions TrollM as a way to support the GABA system, which promotes parasympathetic tone and reduces stress-driven visceral fat storage. He clarifies that this is not a sponsored mention, just a product he recommends.

There's a product called Troscriptions. There's a product called TrollM. Okay, this is using GABA, nicotenol GABA. It's using cava. It's using a couple other compounds to help kind of influence the GABA system.

Also said
“This isn't a sponsor for them, but a product called Troscriptions.”— Discloses no financial relationship.
Find Troscriptions

Glutamine

Supplement

An amino acid that supports GABA production and gut health, recommended through diet and occasional supplementation.

DeLauer learned from Dr. Scott Sure that having ample glutamine available helps produce more GABA, the brain's primary inhibitory neurotransmitter. This promotes relaxation and counters the stress response that drives visceral fat storage. He also notes glutamine's role in gut health, helping to maintain the intestinal barrier and reduce inflammation.

Having a good amount of glutamine coming in the diet through diet and also occasional supplementation can improve how much glutamine you have on hand which actually supports the GABA scale in your body.

Also said
“By having more glutamine available you can produce more GABA. GABA is the parasympathetic tone within your brain. It's the relaxed state.”— Explains the mechanism linking glutamine to stress reduction.
Find Glutamine

Dr. Van Watson's Book on C15

Book

A book by one of the pioneering researchers on C15, recommended for deeper understanding of this fatty acid.

DeLauer mentions that he linked to Dr. Van Watson's book in the description for those who want to learn more about C15 and its role in human health. He does not provide the title but positions it as a resource for further reading.

I also linked out to Dr. Van Watson's book who's one of the pioneering researchers of C15 as this fat that we're deficient in uh across the human race really.

Find Dr.

MOTS-c Peptide (Consult Doctor)

Practice

An exercise mimetic peptide that may enhance fat oxidation; DeLauer would personally talk to his doctor about it if he had significant visceral fat to lose.

DeLauer describes MOTS-c as an exercise mimetic that shows promise in oxidizing fat. He has done detailed videos on it and suggests that if he were in a situation requiring aggressive visceral fat loss, he would discuss it with a knowledgeable physician. He emphasizes that this is for educational purposes only and requires medical supervision.

I would go to my doctor and I would talk to them about something called the MOTS-c peptide. It is an exercise mimetic that seems to have promising effects when it comes to oxidizing fat.

Also said
“This video is for educational and entertainment purposes only. So you need to talk to a practicing physician about this if it's something that would work.”— Standard disclaimer.
Find MOTS-c
Disclosed sponsorships1speaker disclosed

Fatty15

Supplement Sponsored · disclosed

A purified C15 saturated fat supplement that protects liver cells from ferroptosis and reduces liver fat.

DisclosureDeLauer provides a 15% off discount link in the video description and mentions having interviewed the researcher behind it.

DeLauer recommends Fatty15 as an alternative to eating aged cheese for C15 intake. He notes that he has had the researcher on his channel and that the supplement is a convenient way to get the benefits of C15 without the extra calories or dairy. He provides a discount code and links to Dr. Van Watson's book for further reading.

vs alternatives

Compared to dietary cheese, Fatty15 provides isolated C15 without additional calories or lactose, but cheese also offers other nutrients. DeLauer says either is fine.

Fatty 15 is the isolated form of the C-15 saturated fat. So, it makes the cell have structure so it doesn't become oxidized and ultimately lead to premature cellular death within the liver.

Also said
“I put a link down below. That is a 15% off discount link when you use that link and that code down below.”— Discloses affiliate relationship.
Find Fatty15

Notable quotes

Lines worth pulling out — contrarian, specific, or perfectly phrased

6 items
Visceral fat is more than likely an additional proxy for liver fat. What does that mean? It means that it's likely starting as a fatty liver and it's expanding around our organs.
Reframes visceral fat as a downstream consequence of fatty liver, shifting the intervention target.
I don't even care at first if we change our calorie count. Okay? Increase the time between meals and sporadically have longer periods of time.
Challenges the calorie-counting dogma by prioritizing meal timing for insulin sensitivity.
When you have high amounts of stress and cortisol, you're going to have four times as many cortisol receptors in the visceral fat, which means that insulin and cortisol, all these things are going to have a stronger impact on the visceral fat than they would anywhere else in the body.
Quantifies the cortisol-visceral fat connection with a specific receptor density figure.
We need the visceral fat to be burned and activated through the stress response of the high-intensity activity, but we need the metabolic shift of training the liver to oxidize fats and upregulating what's called PPAR.
Succinctly captures the dual-training rationale.
There is now evidence that supports that drinking water can increase the resting energy expenditure for up to 25% up to 25% for up to 60 minutes after consumption.
Puts a surprisingly high number on water-induced thermogenesis, making hydration a metabolic tool.
The more stressed you are, the more likely you are to store fat in that visceral region itself. Okay? The body's just going to reallocate because you have more glucocorticoid receptors there.
Memorable phrasing of stress-driven fat partitioning.

Sign in to share feedback

Tell us if this brief hit the mark or missed it — feedback feeds back into the next iteration of the prompt.

Topics covered

visceral-fatfatty-liverinsulin-resistancemeal-frequencyintermittent-fastingnorwegian-4x4vo2-maxhigh-intensity-interval-trainingzone-2-cardioruckingppar-alphaomega-3-fatty-acidsc15-saturated-fatferroptosisfatty15aged-cheeseglp-1protein-intakemots-c-peptideyohimbine
Free account

Make this library yours

Reading is free for everyone. A free account adds the personal layer: save protocols, follow experts, and see how the other experts weigh in on this same topic.

Create a free accountSign in

Where the experts disagree — weekly

One email a week: the sharpest new disagreements and protocols from the library. No spam, unsubscribe anytime.

Educational summary of the cited expert source — not medical advice. Open the source recording linked above and consult a qualified physician before acting on any protocol.