A fasting insulin test ($25–$35 out of pocket) reveals insulin resistance decades before glucose or A1c become abnormal, giving a 20‑year head start on preventing diabetes, heart disease, cancer, and dementia.
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Ideal fasting insulin is 2–5 mU/L. The standard lab range (2.5–25) is dangerously broad, and 70–80% of US adults are outside the optimal range, making them “silently” insulin resistant.
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High insulin—not high cholesterol or blood sugar—is the key driver of cardiovascular disease, kidney failure, cancer, dementia, obesity, and osteoarthritis through inflammation, sodium/water retention, immune suppression, and mitogenic signaling.
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Intermittent fasting and a low‑carbohydrate diet lower insulin, unlocking hormonally trapped fat stores and breaking the hunger cycle; calorie‑counting fails because it ignores this hormonal lock.
Protocols
Concrete recipes — what, when, how much, and why
4 items
Get a fasting insulin test
WhatMeasure fasting serum insulin to assess early insulin resistance.
WhenAt any routine blood draw; ideally as part of an annual check‑up or when concerned about metabolic health.
DoseA single blood sample after 8–12 hours fasting; cost $25–$35 out of pocket.
For whomAnyone, especially those with family history of diabetes, stubborn weight, PCOS, or unexplained health issues; speaker recommends it for all.
WhyDetects insulin resistance decades before glucose or A1c rise, enabling early intervention to prevent diabetes, CVD, cancer, dementia, etc.
CaveatsStandard lab range (2.5–25) is misleading; target 2–5. If doctor refuses, pay out of pocket or find a cooperative physician.
He argues that this test is the single most undervalued tool in preventive medicine. Because insulin is the body’s ‘key’ that lets glucose into cells, measuring it reveals how hard the pancreas is working to keep glucose normal. He shares that hundreds of his audience members have been denied the test or told it’s useless, reflecting a systemic blind spot. He sees it as a cheap, actionable marker that could slash the $5.6 trillion US healthcare bill if widely adopted.
Mechanism
Insulin resistance forces the pancreas to secrete more insulin to maintain glucose control. Fasting insulin directly quantifies this compensatory hyperinsulinemia.
Personal experience
“I’ve had hundreds of people over the years who have gone to ask for their test and they’re met with exactly these reactions [refusal or anger].”
“you can walk into a lab and pay out of pocket $25 to $35 to get it done.”
Also said
““And if we instead measured insulin, a very simple, inexpensive test, then we would have been able to follow this progress and see that it’s almost a linear progression and we would have been able to catch it before it ever became a problem.””— Emphasizes the early warning power of insulin vs. glucose.
““What you do instead is you find a doctor that is actually interested in your health and understand what we’re talking about here and or you can go and pay for it yourself.””— Advice for overcoming doctor refusal.
Practice intermittent fasting
WhatExtend the daily fasting window (e.g., skip breakfast, eat within a 6‑8 hour window) to allow insulin to drop.
WhenDaily; adjust timing to fit lifestyle.
DoseNo specific fasting duration prescribed; the goal is to go long enough that insulin falls to baseline (e.g., 12–16 hours).
For whomAnyone with elevated insulin or signs of insulin resistance; safe for most but consult a doctor if on medication.
WhyInsulin spikes every time you eat. Frequent eating keeps insulin high all day, driving resistance. Intermittent fasting lowers insulin by creating longer non‑eating periods, which ‘lifts the lock’ on fat stores and reduces hunger.
CaveatsNot a license to overeat in the eating window; best combined with a low‑carb whole‑food diet.
He contrasts ancestral eating patterns (1–2 meals per day, infrequent insulin spikes) with today’s constant eating (3 meals + snacks + sugary drinks, 10–20 insulin spikes). This overload overwhelms cells, causing insulin resistance. Intermittent fasting mimics the pattern our bodies evolved for, giving cells a break from insulin. He says longer gaps between meals significantly drop insulin, allowing the body to tap into stored fat and break the hunger cycle.
Mechanism
During fasting, blood insulin falls, removing the inhibition on hormone‑sensitive lipase, enabling fat cells to release stored fatty acids. Reduced insulin also downregulates fat storage and lipogenesis.
“When with intermittent fasting, when you go longer between meals, now insulin drops significantly when you’re not eating.”
Also said
““So that allows your body to address the real problem.””— Confirms that the benefit is lowering insulin, not just calorie restriction.
““they actually lower insulin.””— States the mechanism simply.
Adopt a low‑carbohydrate diet
WhatReduce intake of sugars and refined carbohydrates to keep post‑meal insulin spikes low.
WhenAt each meal; emphasize protein, healthy fats, and non‑starchy vegetables.
DoseNo specific carb gram target; the aim is to avoid high glycemic foods that spike insulin.
For whomThose with high fasting insulin, obesity, PCOS, NAFLD, or metabolic syndrome.
WhyCarbohydrates, especially sugar, are the primary driver of insulin secretion. Lowering carb intake reduces the total insulin load, helping reverse insulin resistance and allowing fat burning.
CaveatsQuality matters: avoid processed low‑carb products; focus on whole foods.
He explains that the combination of sugar and insulin creates fat, especially in the liver. A low‑carb diet minimizes insulin spikes after meals, which not only reduces fat storage but also allows the body to access stored fat for energy. He notes that this approach works because it targets the underlying hormonal problem, not just calories.
Mechanism
Lower carbohydrate intake reduces the glucose load requiring insulin to transport into cells; less insulin signaling means reduced de novo lipogenesis in the liver and greater lipolysis in adipose tissue.
“And when you’re eating low carb, now it doesn’t rise as much.”
Also said
““And this is why intermittent fasting and low carb diets actually work because they work on the real mechanism. They actually lower insulin.””— Links both dietary strategies to the core problem—insulin reduction.
Find a doctor or pay out of pocket for insulin testing
WhatIf your doctor refuses a fasting insulin test, seek a healthcare provider who understands insulin resistance or order directly from a lab.
WhenWhen your current physician dismisses the request.
For whomAnyone whose doctor won’t cooperate.
WhyMany doctors are resistant to ordering the test; self‑ordering ensures you get the data to guide your metabolic health.
CaveatsBe prepared to interpret results yourself or with a knowledgeable practitioner; see video for ideal range.
Personal experience
“I’ve had hundreds of people over the years who have gone to ask for their test and they’re met with exactly these reactions.”
“What you do instead is you find a doctor that is actually interested in your health and understand what we’re talking about here and or you can go and pay for it yourself.”
What's new
Personal practice updates, fresh positions, predictions
5 items
fasting-insulin-ignored-test
Fasting insulin is the single most informative test for metabolic health, yet doctors refuse to check it or label it useless.
Why this matters: He claims it outperforms glucose, A1c, and cholesterol markers for predicting multiple chronic diseases and that its absence wastes decades of preventive opportunity.
Background
Standard annual checkups measure only glucose and A1c, missing rising insulin for years. The body compensates by pumping out more insulin to keep glucose normal, so the problem remains hidden until the pancreas fails.
He walks through the typical scenario: glucose stays within normal range (65–99) while insulin progressively doubles over years, keeping glucose controlled. A patient can appear “fine” for 20 years, then suddenly be flagged as pre‑diabetic or diabetic—the entire window for reversal has been missed. He highlights widespread doctor resistance, noting that hundreds of his viewers have been met with anger or dismissal when asking for the test. He contrasts the $5.6 trillion US healthcare spending with the $25–$35 cost of the test, arguing that widespread insulin screening could cut the bill dramatically.
Personal experience
“I’ve had hundreds of people over the years who have gone to ask for their test and they’re met with exactly these reactions [refusal or anger].”
“you can walk into a lab and pay out of pocket $25 to $35 to get it done.”
Also said
““But what if they had spent just a few dollars on an insulin test? Then they could actually have saved millions of people with spending very, very little money.””— He frames the missed opportunity in economic terms—a cheap test that could prevent massive healthcare costs and suffering.
““I’ve had hundreds of people over the years who have gone to ask for their test and they’re met with exactly these reactions.””— Anecdotal evidence that the medical establishment routinely blocks the test.
glucose-false-reassurance
Standard glucose and A1c tests remain normal for decades while insulin resistance silently progresses, giving false reassurance.
Why this matters: He provides a concrete numerical progression (glucose 85 → 90 → 95 → 99 → 101 → 126 over years), showing that glucose only flags abnormality after metabolic failure, wasting up to 20 years of early intervention.
Background
Doctors rely on glucose and A1c as primary diabetes markers, but insulin is the upstream controller. As insulin resistance grows, the pancreas compensates by producing more insulin, so glucose stays normal until the pancreas can’t keep up.
He explains that glucose is a tightly controlled variable; the body prioritizes its regulation. Insulin can double or triple before glucose drifts outside the normal range. He gives an example: starting with glucose 85 and insulin 3, over years glucose creeps to 99 while insulin soars, but no red flags appear. Only when glucose hits 101 does a pre‑diabetes flag appear, and within 4–5 years full‑blown diabetes (126) occurs. This means that by the time glucose is high, insulin resistance has been damaging blood vessels, organs, and metabolism for decades. He argues that measuring glucose alone is ‘useless’ for early detection and that insulin testing would catch the problem when it’s reversible. He further notes that cardiovascular disease, cancer, dementia, and obesity are caused by insulin, not blood glucose, so glucose monitoring misses the root cause.
“Glucose is the control variable. So in essence, it looks like nothing changed, but you just became twice as insulin resistant.”
Also said
““And if we instead measured insulin, a very simple, inexpensive test, then we would have been able to follow this progress and see that it’s almost a linear progression and we would have been able to catch it before it ever became a problem.””— Emphasizes that the insulin number would show a clear, early warning trend.
““Cardiovascular disease, cancer, dementia, obesity, and osteoarthritis are caused by insulin, not by blood glucose.””— Directly states that the true damaging factor is insulin, making glucose a useless surrogate.
insulin-causes-most-chronic-disease
Insulin resistance, not high blood sugar, drives heart disease, cancer, dementia, osteoarthritis, kidney failure, PCOS, NAFLD, and obesity through multiple pathways.
Why this matters: He systematically explains distinct mechanisms—inflammation, sodium/water retention, endothelial damage, clotting, immune suppression, mitogenic signaling, brain starvation, and hormonal disruption—tying each to insulin, not glucose or cholesterol.
Background
Mainstream medicine often treats these diseases as separate entities, missing the common root of insulin dysfunction.
He details the mechanisms: (1) High insulin increases sodium reabsorption in kidneys, raising blood pressure, damaging vessel walls and kidneys, leading to kidney failure. (2) Chronic inflammation from high insulin directly damages endothelium, causing the body to send LDL cholesterol to patch rough spots, which over time forms plaques that can rupture (heart attack, stroke). (3) Insulin elevates fibrinogen and clotting factors, making blood stickier and promoting clots that cause heart attacks and strokes. (4) Insulin is mitogenic—speeding cell division—and suppresses immune surveillance (NK cells, T cells), increasing cancer initiation and metastasis, especially breast, colon, pancreatic. (5) In the brain, insulin resistance makes neurons deaf to insulin, starving them of glucose despite high blood sugar, leading to degeneration; it also increases beta‑amyloid plaques. (6) Osteoarthritis is worsened by chemical inflammation changing joint fluid chemistry and by obesity adding mechanical load. (7) PCOS arises from excess insulin raising testosterone, disrupting female hormones and fertility. (8) NAFLD is driven by de novo lipogenesis from sugar and insulin, creating fat in liver and fueling further inflammation and insulin resistance.
“And this is why insulin is a much stronger risk factor for heart disease than cholesterol ever is.”
Also said
““High insulin tells the kidneys to increase sodium retention. … So now with more sodium and more water, we’re going to increase the fluid volume … increase the blood pressure … damage the blood vessels … number one cause of kidney failure and dialysis.””— Illustrates the kidney–blood pressure axis driven by insulin.
““insulin resistance makes the brain deaf to insulin … brain cells will starve despite the fact that the blood glucose … is very high, but inside the cells it can be very low.””— Clarifies the starvation‑in‑plenty paradox in Alzheimer’s.
obesity-hormonal-lock
Obesity is a hormonal condition where high insulin locks fat stores and drives hunger, making calorie counting ineffective.
Why this matters: He reframes obesity away from character flaws, describing a metabolic trap: insulin stores fat and prevents access to it, causing an energy deficit that forces overeating.
Background
Conventional weight loss advice focuses on calorie reduction, ignoring the hormonal mechanisms that control fat storage and appetite.
He likens high insulin to putting money in a vault and throwing away the key—every day, fat is stored and locked away. Because the body cannot access its stored energy, it signals hunger to get more fuel, creating a vicious cycle: eat more → more insulin → more storage/lock → more hunger. This makes calorie‑counting diets fail long‑term; the body fights against the deficit because the fat reserves are hormonally inaccessible. He argues that intermittent fasting and low‑carb diets succeed because they lower insulin, lifting the lock and allowing the body to burn stored fat.
“it’s a hormone that dictates your behavior.”
Also said
““high insulin actually makes you hungry.””— Directly links the hormone to appetite.
““your fat stores, your fat reserves, your energy reserves are hormonally locked down.””— Cement the idea of an inaccessible energy vault.
““And this is why calorie counting diets do not work. … your fat stores … are hormonally locked down.””— Ties the lock mechanism to the failure of conventional weight‑loss methods.
insulin-ideal-range
Ideal fasting insulin is 2–5 mU/L; the typical lab range of 2.5–25 is dangerously broad, and 70–80% of US adults exceed 5.
Why this matters: He quantifies the scale of the problem, asserting that most people have suboptimal insulin levels—a silent epidemic.
Background
Lab reference ranges are based on population averages, not optimal health; a 10‑fold range means many unhealthy individuals are considered ‘normal’.
“you want that number to be between two and five. That’s ideal. … 70 to 80% of them in the US are going to be above that.”
Also said
““There is absolutely no way that 10 times as much insulin as 2.5 would be normal or okay.””— He undermines the legitimacy of the standard lab range.
Disclosed sponsorships3speaker disclosed
Sten Ekberg’s fasting insulin test (direct‑to‑consumer)
Service Sponsored · disclosed
He offers a $25–$35 fasting insulin test that listeners can order without a doctor, as an alternative to physician refusal.
DisclosureSpeaker promotes his own lab testing service; a link to order the test through his company is provided in the video description.
He positions this test as a simple, accessible way to obtain the crucial insulin number. He notes that hundreds of people have been denied the test by their doctors, so his service removes the gatekeeper. He contrasts the trivial cost with the massive disease and economic burden of undetected insulin resistance, emphasizing that for a few dollars one can get a 20‑year head start on prevention.
vs alternatives
Compared to traditional lab orders through a doctor, his service eliminates gatekeeping and allows anyone to self‑order without a prescription.
“I’m going to put a link down below if you want to order it from us.”
Also said
““$25 to $35””— Confirms the low price point.
““or you can just go to one of these places where you can walk in the door and give them a few dollars.””— Notes the accessibility of direct lab testing.
Comprehensive panel ($387) with NMR lipoprotein analysis
Service Sponsored · disclosed
A broader blood panel covering 60‑70 markers, including detailed cholesterol size measurement (NMR) and heart disease risk indicators.
DisclosureSame as above; this expanded panel is also offered by his company.
“we also have a comprehensive panel for $387 where you get 60 70 different markers. You get very detailed markers for cholesterol and heart disease risk. We measure the size of the cholesterol. We give you an NMR analysis and lots of other good stuff.”
Lines worth pulling out — contrarian, specific, or perfectly phrased
6 items
“There is one simple, very inexpensive test that can tell you more about your risk of heart disease, diabetes, cancer, and dementia than any other test.”
Bold opening claim that sets the stage for the entire video.
“But what if they had spent just a few dollars on an insulin test? Then they could actually have saved millions of people with spending very, very little money.”
Contrasts the trillion‑dollar war on cancer with the negligible cost of an insulin test to highlight a massive missed opportunity.
“70 to 80% of them in the US are going to be above that [insulin >5].”
Quantifies the silent epidemic, making insulin resistance a near‑universal concern.
“And this is why calorie counting diets do not work. … your fat stores, your fat reserves, your energy reserves are hormonally locked down.”
A direct challenge to conventional weight‑loss wisdom, arguing that the problem is hormonal, not caloric.
“insulin is a much stronger risk factor for heart disease than cholesterol ever is.”
Controversial statement that demotes the role of cholesterol while elevating insulin as the true culprit.
“Well, actually, if you’re asking me, I think it could have some value. But then again, that’s just me.”
Sarcastic finale after repeatedly asking ‘would that be useless?’, underscoring his frustration with medical dismissal.
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Educational summary of the cited expert source — not medical advice. Open the source recording linked above and consult a qualified physician before acting on any protocol.